منابع مشابه
The f3-LipoproteinDoubletin Type 3 Hyperlipoproteinemia
We used a previously reported agarose-gel electrophoresis system to study the lipoprotein patterns in patients with type 3 hyperlipoproteinemia. All samples from 20 subjects tested revealed the presence of two bands with beta-lipoprotein mobility, irrespective of quantitative changes in plasma lipids induced by diet or by the administration of drugs. If these observations are confirmed, the met...
متن کاملType IV hyperlipoproteinemia in a consanguinous family.
The involvement of all five offspring of consanguinous and hypertriglyceridemic parents (first cousins) is reported. Three sons and two daughters of this family have increased triglycerides, normal cholesterol, and normal or slightly increased phospholipids. Mother and elder daughter have abnormal glucose tolerance. Father and mother are hypertensive; father had recent development of myocardial...
متن کاملType II hyperlipoproteinemia in mother and twins.
A 36-year-old housewife with type II hyperlipoproteinemia became pregnant with twins. Although the pregnancy was uneventful, the serum triglycerides were elevated during the third trimester, the delivery, and early puerperium. The mother had no complications directly related to the pregnancy despite hypercholesterolemia, coronary atherosclerosis, and hypertension. Cord blood cholesterol and bet...
متن کاملHyperlipoproteinemia with albumin-lipid complex: a novel type of hyperlipoproteinemia associated with insulin-resistant diabetes mellitus.
We describe a new phenotype of hyperlipoproteinemia in two members of a family with a high degree of consanguinity. Both have a history of uncontrolled diabetes mellitus without ketoacidosis, and a family history of coronary artery disease at a relatively early age. A high degree of insulin resistance was found. The abnormal lipoprotein(s) has alpha-lipoprotein mobility on cellulose acetate ele...
متن کامل830 Jenwitheesuk
derived from the heptad repeat 2 (HR2) region of HIV1 glycoprotein 41 (gp41). The inhibitor binds to the gp41 heptad repeat 1 (HR1) region, thereby blocking viral HR1/HR2 association. Mutations in HR1 have been reported to cause enfuvirtide resistance and reduce viral fitness. In this study, we first showed that scores obtained by a residue-specific all-atom probability discriminatory function ...
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ژورنال
عنوان ژورنال: Pediatric Research
سال: 1978
ISSN: 0031-3998,1530-0447
DOI: 10.1203/00006450-197804001-00835